UPA

Melanotan 2 (also referred to as Melanotan II) is a synthetically produced variant of a peptide hormone naturally produced in the body that stimulates melanogenesis, a process responsible for pigmentation of the skin. This peptide hormone, called alpha-Melanocyte stimulating hormone or MSH, activates certain melanocortin receptors in the process of exerting its effects. Indeed, MSH also exerts potent influence over lipid metabolism, appetite, and sexual libido via these melanocortin receptors. As a result, Melanotan 2 has been shown in studies to exhibit appetite suppressant, lipolytic, and libido-enhancing effects in addition to promoting skin tanning. Melanotan 2 has been researched extensively for use in protecting against the harmful effects of ultraviolet radiation from sunlight due to its melanogenesis-stimulating properties.

GW501516 (Cardarine) doesn’t have any published human studies. In rodents, though, it shows great promise as an exercise mimetic – it lights up many of the same genes you’d activate by going on a run or lifting [4]. That alone wasn’t enough to make much of a change, but when researchers had mice take GW501516 with consistent exercise, results went through the roof:

Oxandrolone was often refereed to as an all purpose oral AAS. This drug was once marketed under the product name (still commonly used trade name) of Anavar. It has the unique quality of significantly stimulating (more than other AAS) the synthesis of phosphocreatine in muscle cells which in turn provides faster regeneration of, and a distinct elevation in, ATP. Of course all AAS have this effect to some extent. Oxandrolone is simply unmatched in this aspect.

Andarine has been one of the most widely tested and studied of all the SARMS. Tests have proven that Andarine helps retain lean muscle mass as well as enhancing it. However with Andarine this is not the only noted benefit. In addition to strength and lean muscle gains is its powerful fat burning properties as well.

Healthy men who took LGD-4033 for 21 days saw a significant increase in lean body mass. The only side effect was short-term testosterone suppression (more on that in a second) [3]. One milligram per day was enough to cause significant muscle growth. The higher the dose, the more muscle participants put on. In another trial, participants took doses as high as 22 mg/day with no side effects or safety issues [3].

CJC-1295 DAC has shown some amazing results as a growth hormone releasing hormone (GHRH) analog. Not only has CJC-1295 shown the ability to increase growth hormone and IGF-I secretion and its benefits, but it has been able to do so in very large amounts. Recent research studies have shown that CJC – 1295 stimulates GH and IGF-1 Secretion, and will keep a steady increase of HGH and IGF-1 with no increase in prolactin, leading to intense fat loss, and increases protein synthesis. CJC-1295 DAC is a long acting Growth Hormone Releasing Hormone, which causes the anterior pituitary to release more growth hormone. GHRH is released in pulses in the body, which alternate with corresponding pulses of somatostatin (growth-hormone inhibiting-hormone). Clinical Research was first conducted for CJC-1295 during the mid-2000s. The objective of the peptide was to treat visceral fat deposits in obese AIDS patients, as increased levels of exogenous hgH are presumed to increase lipolysis (fat loss). The clinical research was ultimately successful for most research subjects. Ghrelin, released from the gut, which circulates and acts as a hunger hormone, has synergistic activity in the body with GHRH and also suppresses somatostatin to make way for the GHRH pulse. Studies shows that combining a GHRP-6 with CJC 1295 DAC, significantly increase the release of GH and IGF-1 production without an increase in prolactin. An example of a GHRP (GH Releasing Peptide) is Hexarelin or GHRP-2. CJC 1295 DAC is a exceptionally designed peptide and is known for being the finest of the hGH secretogues. The DAC (Drug Affinity Complex) portion increases the half-life by binding with serum albumin and protects the CJC-1295 DAC peptide from degradation. This was formed when a lysine link was bounded to DACs to a reactive chemical called maleimidoproprionic acid (MPA). CJC-1295 DAC vs. CJC-1295 No DAC